CMAJ • June 20, 2006; 174 (13). doi:10.1503/cmaj.051671.
© 2006 CMA Media Inc. or its licensors
All editorial matter in CMAJ represents the opinions of the authors and not necessarily those of the Canadian Medical Association.
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Sudden infant death syndrome

Carl E. Hunt and Fern R. Hauck

From the National Heart, Lung, and Blood Institute (Hunt), Bethesda, Md., and the Departments of Family Medicine and Public Health Sciences, University of Virginia (Hauck), Charlottesville, Va.


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  		Fig. 1: Schematic summary of potential interactions between environmental and genetic risk factors for sudden unexpected death in infancy and sudden infant death syndrome (SIDS). The clinical consequences (phenotype) are not known for the autonomic nervous system and serotonin transporter (5-HTT) polymorphisms. Adapted from Hunt.70

 

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